Hi! great job. Could you elaborate a little more on what your species is? I am also curious to know what implications this has in the world of science, beyond what is going on inside the cell!
Hi Rebecca! so the species I worked with was Tetrahymena thermophila which are cilates. They are these tiny little things that are super cute and can be seen with a microscope. They’re important to my research because of how they cause double stranded breaks in their own genome every time they mate and then repair each of those breaks. This tells me that they are very good at repairing DNA and have a process of set proteins to do so.
The implications of my research in science as a whole is all about cancer treatments. The DNA has to be repaired and understanding the different ways in which the cell can repair DNA can help make drugs that either prevent or help treat cancer. However, my specific results suggest that my gene is not applicable to that which is also important to know. It will take more research but knowing something is not involved is helpful as well.
Hi Nicholas! I also watched your presentation. Amazing job!!
My gene has homologs in humans as well as Mus Musculus. The human homologs are most important to us because that means if this gene had been involved in DNA damage repair then it would have suggested that the homolog in humans does as well and could be a potential target of future cancer treatments. However, if my results are validated in the future, my gene was not thought to be involved in DDR so probably won’t be used as a target for such treatments in the future.
Hi! great job. Could you elaborate a little more on what your species is? I am also curious to know what implications this has in the world of science, beyond what is going on inside the cell!
Hi Rebecca! so the species I worked with was Tetrahymena thermophila which are cilates. They are these tiny little things that are super cute and can be seen with a microscope. They’re important to my research because of how they cause double stranded breaks in their own genome every time they mate and then repair each of those breaks. This tells me that they are very good at repairing DNA and have a process of set proteins to do so.
The implications of my research in science as a whole is all about cancer treatments. The DNA has to be repaired and understanding the different ways in which the cell can repair DNA can help make drugs that either prevent or help treat cancer. However, my specific results suggest that my gene is not applicable to that which is also important to know. It will take more research but knowing something is not involved is helpful as well.
Thanks! that makes sense.
I really liked the presentation! I was wondering, is this gene present in other cells besides this one?
Hi Nicholas! I also watched your presentation. Amazing job!!
My gene has homologs in humans as well as Mus Musculus. The human homologs are most important to us because that means if this gene had been involved in DNA damage repair then it would have suggested that the homolog in humans does as well and could be a potential target of future cancer treatments. However, if my results are validated in the future, my gene was not thought to be involved in DDR so probably won’t be used as a target for such treatments in the future.