Is there a reason you chose to use Berbine Chloride as the antibiotic? I think you did a really good job at explaining the differences and reasons you chose the bacteria that you did and you also thoroughly explaining the procedures in a way for most people to understand.
Sorry about asking the same question I typed it all out and refreshed and someone had asked the same thing. I can’t delete the above comment so I will ask another question below.
It’s cool that your experimental results supported your hypothesis! I am curious as to why you said that the medication would need to be tested specifically on media simulating liver cells, gut cells, and neurons? I know you said to make sure it was safe on those media but I wonder why those specific media were picked.
It is cool that your experimental results matched your hypothesis! I was wondering why the medication would need to be tested specifically on media simulating liver cells, gut cells, and neurons? I know you said to make sure it is safe for those media but why did you pick those media specifically?
the liver cells, gut cells, and neurons were just examples of future directions and don’t necessarily have any experimental priority over other types of cells
In your first graph when you say that the two concentrations below the HIT line would be a “viable dosage” for the antibiotic, does this mean it could possibly be used as a treatment in humans? What is the dosage referring to?
What made you choose the Berberine Chloride as an antibiotic for Salmonella? Do the infections it typically fights have a similar bacteria?
my colleague has experience using the Goldenseal Herb as traditional medicine, which contains Berberine.
Is there a reason you chose to use Berbine Chloride as the antibiotic? I think you did a really good job at explaining the differences and reasons you chose the bacteria that you did and you also thoroughly explaining the procedures in a way for most people to understand.
Sorry about asking the same question I typed it all out and refreshed and someone had asked the same thing. I can’t delete the above comment so I will ask another question below.
It’s cool that your experimental results supported your hypothesis! I am curious as to why you said that the medication would need to be tested specifically on media simulating liver cells, gut cells, and neurons? I know you said to make sure it was safe on those media but I wonder why those specific media were picked.
Apologies for asking the same question after I posted I reloaded and it was the same I will ask another question below.
It is cool that your experimental results matched your hypothesis! I was wondering why the medication would need to be tested specifically on media simulating liver cells, gut cells, and neurons? I know you said to make sure it is safe for those media but why did you pick those media specifically?
the liver cells, gut cells, and neurons were just examples of future directions and don’t necessarily have any experimental priority over other types of cells
In your first graph when you say that the two concentrations below the HIT line would be a “viable dosage” for the antibiotic, does this mean it could possibly be used as a treatment in humans? What is the dosage referring to?
the concentrations that were below the HIT line had at least a statistically 95% success rate of using Berberine to kill the bacteria.