Hello! I really enjoyed listening to your presentation. You mentioned the goal of determining the specific role and essentiality of Rdn2 by knocking out the gene and observing the effect. Is there a particular laboratory method or procedure that you recommend that would allow you to accomplish this? Great job!
While I’m not extensively familiar with gene editing, CRIPSR-Cas would be able to take out the gene and leave the rest of the cell relatively untouched and we would see what happens 🙂
Thank you so much for watching!
I really liked your presentation, it was super interesting! Why would you want to see what Rdn2 did and to what extent it did it at? What would doing those experiments help you find?
If the DNA repair mechanism is conserved across eukaryotes, there is a high likelihood that we have the same pathway in our cells. This could be exploited to create more cancer treatments, and much more!
I’m glad you enjoyed it!
Great presentation! In the future directions you mention isolating the expression of Rdn2. What benefits would you find in determining the conservative pathways within single celled eukaryotes and how could the knowledge be utilized?
Nice presentation! With Rdn2 tripling when DNA is damaged, does this speed up the process of repairing or start the repair process? If it speeds up the process, is there a way to increase the amount of Rdn2 produced to make the DNA repair even quicker?
We actually don’t know! Its presence indicates that it is involved with the DNA repair pathway, but more experiments would be needed to figure out what it actually does. There might be additional literature out there that has more answers, but I have not yet found it
If DNA goes untreated, mutations continue to arise which leaves the potential for issues, especially cancer. I’m glad you enjoyed my presentation! 🙂
curesymposium.org 
Hello! I really enjoyed listening to your presentation. You mentioned the goal of determining the specific role and essentiality of Rdn2 by knocking out the gene and observing the effect. Is there a particular laboratory method or procedure that you recommend that would allow you to accomplish this? Great job!
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While I’m not extensively familiar with gene editing, CRIPSR-Cas would be able to take out the gene and leave the rest of the cell relatively untouched and we would see what happens 🙂
Thank you so much for watching!
LikeLike
I really liked your presentation, it was super interesting! Why would you want to see what Rdn2 did and to what extent it did it at? What would doing those experiments help you find?
LikeLike
If the DNA repair mechanism is conserved across eukaryotes, there is a high likelihood that we have the same pathway in our cells. This could be exploited to create more cancer treatments, and much more!
I’m glad you enjoyed it!
LikeLike
Great presentation! In the future directions you mention isolating the expression of Rdn2. What benefits would you find in determining the conservative pathways within single celled eukaryotes and how could the knowledge be utilized?
LikeLike
I loved the presentation and project! Could you explain some of the limitations of your project a bit more clearly for me?
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Nice presentation! With Rdn2 tripling when DNA is damaged, does this speed up the process of repairing or start the repair process? If it speeds up the process, is there a way to increase the amount of Rdn2 produced to make the DNA repair even quicker?
LikeLike
We actually don’t know! Its presence indicates that it is involved with the DNA repair pathway, but more experiments would be needed to figure out what it actually does. There might be additional literature out there that has more answers, but I have not yet found it
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Awesome presentation! I was wondering what are the potential issues that arise with DNA damage?
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If DNA goes untreated, mutations continue to arise which leaves the potential for issues, especially cancer. I’m glad you enjoyed my presentation! 🙂
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