Is it reasonable to conclude that these findings are applicable in different model organisms such as humans, and what specifically would be the next tests/experiments in the process of potentially applying this finding to human conditions?
Ultimately we cannot conclude just yet that the Twi8 protein can quite be applicable to humans. However, we will use this information for gene knockout, qPCR, and even protein localization to further study these proteins involved in DNA damage repair.
In order to make these results potentially applicable to cancer research we would use this information to perform a qPCR to further understand the role of the Twi8 protein and to measure its expression levels.
Can you explain more about the positive attributes and capabilities you were referring to when explaining why you used T. thermophila as the model organism?
Using T. thermophila as a model organism is very effective in this type of research because it is known for its ability tom perform extensive amounts of DNA damage repair naturally and respond to environmental pressures.
In testing the Twi8 gene in human genomes, would you likely use the same method to induce DNA damage, or would you find a different way to induce this damage? If you do use the same method, how do you expect it would interact with the human genome?
When inducing DNA damage in humans, I can only imagine we would use the same method in Rad51 because it was used as a positive control in this experiment. On the other hand, you could probably take human DNA that is already damaged due to natural stressors like UV light and then test the Twi8 gene efficacy in DNA damage repair processes.
curesymposium.org 
Is it reasonable to conclude that these findings are applicable in different model organisms such as humans, and what specifically would be the next tests/experiments in the process of potentially applying this finding to human conditions?
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Ultimately we cannot conclude just yet that the Twi8 protein can quite be applicable to humans. However, we will use this information for gene knockout, qPCR, and even protein localization to further study these proteins involved in DNA damage repair.
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What would be the most ideal future experiment to perform in order to make these results potentially applicable to cancer research?
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In order to make these results potentially applicable to cancer research we would use this information to perform a qPCR to further understand the role of the Twi8 protein and to measure its expression levels.
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What would be the ideal future experiment to perform in order to make these results potentially applicable to cancer research?
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Can you explain more about the positive attributes and capabilities you were referring to when explaining why you used T. thermophila as the model organism?
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Using T. thermophila as a model organism is very effective in this type of research because it is known for its ability tom perform extensive amounts of DNA damage repair naturally and respond to environmental pressures.
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In testing the Twi8 gene in human genomes, would you likely use the same method to induce DNA damage, or would you find a different way to induce this damage? If you do use the same method, how do you expect it would interact with the human genome?
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When inducing DNA damage in humans, I can only imagine we would use the same method in Rad51 because it was used as a positive control in this experiment. On the other hand, you could probably take human DNA that is already damaged due to natural stressors like UV light and then test the Twi8 gene efficacy in DNA damage repair processes.
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How would the Twi8 gene induce DNA damage in different species?
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